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He was also awarded the college medal for scholastic philosophy. Mono- or polyunsaturated fatty acids MUFA: Lidocaine or longer acting bupivicaine are sometimes given using implanted pumps that deliver tiny quantities to the fluid that bathes the spinal cord, where they can quiet excess firing of pain cells without affecting the rest of the body. The exception is microglia , which are derived from hemopoietic stem cells. Amines are deactivated by the enzyme monoamine oxidase or diamine oxidase, and some antidepressants such as monoamine oxidase inhibitors, inhibiting the enzyme.

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After three years, he was given a clean bill of health by three neurologists in three different places and was given a responsible position. This was in The individual remains in excellent health, but continues with modified therapy.

Male, white, receiving treatment from nearby medical centre for one year. He was receiving guanadine amount unknown and 90 mg. He was first seen in a Myasthenia Gravis crisis. The emergency treatment consisted of two ampules of prostigmine methylsulfate of a strength of 1: Within a period of eight or ten minutes, the patient experienced a generalized convulsive seizure which lasted some five minutes and required 4 men to hold him on the bed. Prostigmine, by needle, was continued for three weeks, and then 15mg.

Thiamin hydrochloride was given three times each day, intramuscularly, as well as other fractions of the B complex. Although given only two weeks to live by the physicians at the medical centre the day prior to our first visit, this individual lived a normal life for 18 years. His death was due to a cerebral accident. Female, white, with diagnosis August , Polyneuritis.

Began with pain and burning of legs associted with jerking. Ran high fever 10 days. Paralysis started on left side along with weakness of hands, soon followed with complete paralysis lower extremities.

Paralysis and weakness as described. Started on medication by mouth and intramuscular injections. Several months later, began intravenous schedule.

In approximately 16 months, was able to move right leg. Upper extremities returned to normal. Patient now able to walk approximately 50 yards with knee braces and walker. Does all the cooking for family of four, as well as sewing clothes for herself and two daughters. I can personally vouch for her ability as a cook. April , she was able to go without a back brace that was previously necessary for her to use to even get out of bed.

Our diagnosis in this case is Transverse Myelitis. She has also received mg ribonucleic acid four times each week. Female, white, who developed weakness in extremities around June 25, Sensory examination revealed hypalgesia over medial aspect of right foot and calf.

Motor examination revealed a partial foot drop on the right, with rather marked weakness and inversion, eversion, and dorsiflexion of right foot. Reflexes upper extremities plus. Knee jerks were plus with patellar clonus.

Right ankle jerk was 4 plus and the left, 3 plus. Bilateral, sustained, ankle clonus. Later examined and hospitalized at a nearby medical centre where Medrol was tried. She was sent home with a diagnosis of Multiple Sclerosis, superimposed by a viral meningoencephalitis.

Blurring of vision was established as due to a left six-nerve paralysis. She came home to a wheelchair provided she lived. Seen in our office one month later, we concurred with the impression of Multple Sclerosis. Our treatment schedule became operative. It has been a long journey since June , but the results have been phenomenal. This individual has been returned to full activities, and as a gesture of gratitude, comes to my office to serve in the capacity of an office assistant several days each week.

She does, however, still maintain her treatment schedule. Whether this is necessary or not, I follow the advice of another patient who has been continuing modified treatment for 22 years: Male, white, 28 years. History of numbness in lower extremities with loss of muscle control from waist down.

This started approximately 2 years before this visit. Difficulty with bladder control at times. Seen by several neurologists at a nearby medical centre who failed to make a diagnosis other than to say he had a central Nervous System Pathology. Ankle clonus was bilateral and sustained on right.

He demonstrated a right foot drop. We entertained a diagnosis of Multiple Sclerosis. We advised him not to accept ACTH therapy. The following week we did start treatment. After 5 weeks, we did not see the patient again for three weeks, at which time he confessed that he thought that he was well and had stopped treatment. The weakness and other symptoms were again returning.

He has been back to gainful employment for the past 12 months. Female, white, 57 years. Chief complaint was fatigue. This started approximately seven years before coming to our office.

The onset of illness was gradual. Generalized weakness as the day went on, but was always feeling refreshed in the morning. Drooping of the eyelids became a problem so that she automatically would tilt her head backward so that the ptosed eyelids would be partially corrected. Fatigue of the muscles of mastication on chewing became so embarrassing that for the past several months, she avoided all social events, even dinner with friends. Swallowing also became a serious problem forcing her to a bland and sometimes liquid diet.

Even a few minutes talking, while taking the history, would so fatigue her that she found it necessary to recline on the examining table so as to regain her strength. She visited many clinics and medical centers in the United States and Europe, but always was given the same diagnosis — her review of conditions labeled her as psychosomatic. To us it was obvious that she suffered from advanced Myasthenia Gravis. Thiamin Hydrochloride and mg.

She remarked that she had not been able to do that in three years. She was given our schedule for treatment, but had great difficulty getting her local physician or any physician to give her the needed injections. In desperation, he returned to one of the medical centres and confronted them with the diagnosis, which they did not believe.

She, however, demanded that they employ their test for this disease, which they did. Her response was the greatest ever seen in that University. She is also receiving RNA mg. She no longer hesitates to eat in public, and her stamina is approaching normal. During a visit to our office in April of , she laughed and joked about her experiences in getting the diagnosis confirmed so that she could receive the vitamin injections under supervision.

She also favored us with a platter of delicious cakes that she had baked. Although we could write a book on cases treated and cured or established a permanent remission , time is a prohibiting factor. The treatment of Multiple Sclerosis has been empiric since it was first described by Sir Robert Carswell in Brickner, 9 in , gave a review on treatment which included preparations of Antimony and Arsenic, fever induced by various methods such as diathermy, malaria, typhoid vacine, and fever brought on with the use of drugs.

Surgical procedures such as cervical sympathectomy and root section were also employed. Serums, hypnotism and intraspinal injections of lecithin had their day. Moore administered nicotinic acid and thiamin following the dissertation by Zimmerman and Burack 10 on diseases of the nervous system resulting from a deficiency of the vitamin B-Complex, and the paper by Spies 11 and others on the use of nicotinic acid in the treatment of Pellagra associated with mental pathology.

Spies and Aring, 12 in , published a paper on the effects of Vitamin B1 on peripheral neuritis as associated with Pellagra. Moore also had the benefit of the work of Stern, who published an article on the intraspinal use of Vitamin B1 for the relief of intractable pain, and for inflammatory and degenerative diseases of the Central Nervous System. We learned early in our approach to this disease that small and infrequent doses of thiamin hydrochloride would not accomplish our purpose, and we also realized that more than one unit of the B-Complex would be required, even though the physiological chemistry relative to this phase of metabolism had not been completely established.

Although Moore used nicotinic acid for vaso-dilation purposes, we rationalized that the degenerative process taking place in nerves, and thus also in muscle, was of a greater magnitude.

Inasmuch as the only sickness remembered by the patient, family or relatives took place during the summer months, 13 we immediately suspected a virus to be the offending agent. This idea gained momentum with the greater incidence of Multiple Sclerosis following the epidemic of encephalitis lethargia of to , and the epidemic of encephalitis B in St. Louis and Toledo in However, the incidence of Polio was also up.

Mixed, abortive or unrecognized cases of Poliomyelitis became a tantalizing factor. After the isolation of the Coxsackie virus with its mimicking of Polio, and the knowledge that the paralysis with this type virus infection was never permanent, the real devastating factor, in time and place, at least to me, became apparent. Flexner and Lewis 14 were able to demonstrate that in Polio, vascular and lymphatic lesions constituted the primary causes of the lesions of the nervous system.

Multiple hemorrhagic accidents take place in Multiple Sclerosis with ensuing scar tissue. As these microscopic scars contract, they impinge on the vessels carrying nutrients to the Central Nervous System cells. For this reason, the Sister Kenney treatment for Polio had merit, since it helps to maintain muscle and muscle-nerve integrity.

Our employment of nicotinic acid is to effect adequate dilatation of existing vascular structures, producing over time, chemically, what the Urologist accomplished with his catheters in a mechanical fashion. Once these channels are sufficiently operative, the metabolic factors that we supply will go about revamping the myelin sheaths. Due to lack of full energy components, cells can temporarily lose the ability of normal physiological activity.

We can restore the normal function of cells which depends upon their ability to extract and use the chemical potential energy locked within the structure of organic molecules. We accomplish this by placing massive amounts of the essential material at the disposal of cells.

We categorically make this statement: Any victim of Multiple Sclerosis who will dramatically flush with the use of nicotinic acid, and who has not yet progressed to the stage of myelin degeneration, as witnessed by sustained ankle clonus elicited in the orthodox manner, can be cured with the adequate employment of Thiamin Hydrochloride and other factors of the Vitamin B Complex in conjunction with essential proteins, lipids, carbohydrates and injectable crude liver.

If sustained ankle clonus is not bilateral, then it is not a deterrent. We have had patients who did demonstrate bilateral sustained ankle clonus, and who were in wheelchairs, and who returned to normal activities after 5 to 8 years of treatment. These patients, fortunately, had not received ACTH. One patient was given a single course of Medrol 4 mg. This had little effect on her pathology, and apparently no blocking action, on our treatment. The general use of ACTH in Multiple Sclerosis will extend the recovery period by a time directly proportional to the amount of the drug employed.

However some oils from tropical plants such as palm oil and coconut oil do contain some saturated fats. The main unsaturated fats are monounsaturated, found particularly in foods such as olive oil, rapeseed oil, peanuts and avocados.

Polyunsaturated fats are mostly found in plant foods such as nuts, seeds and vegetable oils, and in cold-blooded sea-foods. In natural foods, they come protected with antioxidant vitamins. There are two main classes polyunsaturated fatty acids, omega-3 and omega These include the essential fatty acids.

Trans fats can be natural or artificial. They are mostly artificially created through a process known as hydrogenation which involves heating and chemical structure change. Artificial trans fats are mostly found in fast foods, fried foods and commercial baked products such as cookies and are the most unhealthy fats even worse than saturated fats! Natural trans fats can be found in small amounts in milk and beef, and in quite large concentration in cheese.

Why do we need cholesterol? Now, we understand that we do actually need some fat in our diet for survival and in the end, it is all about getting the amount and the right balance! Why do we need to eat fat? Conventional methods of treating pinched nerves usually include medications and surgery.

However, research shows that non-surgical, more conservative treatments, including physical therapy, exercise, chiropractic adjustments , supplements and rest, can also greatly help reduce pinched nerve pain. Pinched nerves also called compressed nerves are deep root nerves that have become inflamed and irritated due to experiencing an abnormal amount of pressure. Nerves are responsible for sending important sensory information regarding pain, well-being and perceived threats from our bodies to our brains, and vice versa.

Major nerves travel from your brain through your spinal cord and down the center of your back, connecting to small series of nerves that stem off into your limbs and elsewhere.

What are some common conditions that might cause a pinched nerve? Compression increased pressure and stress placed on a root nerve is the primary cause of a pinched nerve, which interferes with normal signals regarding pain. There are several locations in the body where pinched nerves are common and numerous reasons that someone might develop a pinched nerve.

The causes of a pinched nerve can include: For the most part, people use the terms herniated disc, bulging disc, slipped disc, and pinched or compressed nerve interchangeably. Herniated discs and slipped discs can contribute to pinch nerves because they cause tissue to protrude into a nearby nerve.

These conditions cause spinal discs to open and expand, which can lead to fluid leaking out, worsened inflammation and increased pressure. Prior to taking medications or receiving adjustments, and definitely before undergoing surgery, getting an accurate diagnoses is crucial. What does a pinched nerve feel like? Pain, nerve damage and irritation caused by a pinched nerve can sometimes be minor but other times severe.

Pain can occur in the cervical neck region, thoracic upper region or lumbar lower spine. Although the location of a pinched nerve determines the types of symptoms you feel, most pinched nerves have the following in common: Increased pain when moving and trouble exercising are also common pinched nerve symptoms.

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